Cause 3: Copper Accumulation – The Silent Invader
The excess copper in Wilson’s disease acts like an uninvited guest, accumulating silently in the body’s tissues, primarily in the liver and brain. This accumulation doesn’t happen overnight; it’s a gradual process, flying under the radar until the damage is done. The liver, being the primary site for copper metabolism, bears the brunt of this toxic overload.
As the copper builds up, the liver starts waving white flags, signaling distress. Inflammation sets in, followed by scarring, and if left unchecked, the liver heads towards failure. It’s a slow burn, with the body’s own detox center struggling to keep up with the copper onslaught.
The brain doesn’t escape unscathed. The basal ganglia, a critical area for controlling movement, becomes a hotspot for copper accumulation. Here, the metal wreaks havoc, disturbing neurological functions and leading to a range of symptoms from tremors to difficulty swallowing. It’s like a storm brewing in the brain, disrupting the calm waters of normal functioning.
Addressing copper accumulation in Wilson’s disease requires a two-pronged approach: reducing the excess copper and preventing further accumulation. Medications like chelating agents step into the ring, binding to the copper and helping the body excrete it. Meanwhile, dietary changes serve as a preventative measure, reducing copper intake and giving the body a fighting chance.
In closing, copper accumulation is the silent force behind Wilson’s disease, a force that demands attention and action. Through early detection, comprehensive treatment, and proactive management, we can keep this invader at bay, preserving health and quality of life. (3)
